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Introduction Brothers and sisters, a few days ago, I have discovered multiple mathematical patterns in surah Baqarah. I have even unraveled the meaning of the mysterious letters at the beginning of the chapter "alm". Before continuing to read this post though, I recommend you learn about the number 19 and it's importance in the Quran if you don't know about it already. Frequency of The Letters "ALM" in Surah Baqarah The frequency of the letters "alif, lam, meem" are very important in understanding the mathematical patterns in this chapter. Alif (أ): Appears in all 286 verses Lam (ل): Appears in all 286 verses Meem (م): Appears in 283 verses The Miraclous Math If we were to add the three numbers listed above (286 + 286 + 283), we will come to a sum of 855. 855 is a multiple of 19 (19 x 45). But I'm not done. 855 / 19 = 45. The sum of all the digits (2 + 8 + 6 + 2 + 8 + 6 + 2 + 8 + 3) in 286;286;283 is... 45. Still not done. If we were to concatenate the three numbers in the order they already are, we would get 286,286,283. We need to add the number 2 to the beginning though as an identifier for surah Baqarah (second chapter in Quran). The final number is 2,286,286,283. That number, is indeed a multiple of 19 (2,286,286,283 / 19 = 120,330,857). And 120330857 is also a multiple of 19 (120330857 / 19 = 6,333,203). Conclusion The Quran is the word of God. Nobody could recite such perfection.
Absolute truth posted a topic in Islamic DiscussionsIntroduction One of the most popular alleged evidences for evolution on the internet is Endogenous RetroViral sequences (ERVs). Evolutionists think that a type of virus called a 'retrovirus', once inserted genetic information into one of our ape ancestors' genome. So how is this evidence for evolution? Scientists have noticed that chimps and humans have ERV genetic sequences at very similar points in our DNA. And so the story goes: our common ancestor acquired these ERVs and since humans and chimps are closely related, we should have them in similar spots in our genomes. We do. If we and chimps didn't evolve from a common ancestor (which first acquired the ERVs), how is it possible that we and chimps have ERVs in almost precisely the same locations? The only plausible explanation, evolutionists say, is evolution. But this is far from the truth. If we can show that ERVs are not the product of retroviruses, this evidence for evolution would fall flat. ERVS are simply more or less similar to retrovirus genome: The first problem with this argument is that it’s hard to tell what an ERV is when you meet one. It doesn’t come with a tag attached saying: ” This is an ERV “. It could be that these genes something completely different. That is because if a virus is embedded in it’s complete form, its almost impossible to pass it down to further generations. ERVS are Functional If ERVs are found to have function, it would be highly likely that they didn't originate from retroviruses. It would be inconceivable that viral non-functional ERVs somehow became functional. Evidence has surfaced that they do have function. "We report the existence of 51,197 ERV-derived promoter sequences that initiate transcription within the human genome, including 1,743 cases where transcription is initiated from ERV sequences that are located in gene proximal promoter or 5' untranslated regions (UTRs)."(Conley, A.B., Piriyapongsa, J. and Jordan, I.K., "Retroviral promoters in the human genome," Bioinformatics 24(14):1563, 2008) The previous quote is very telling. There are many thousands of ERV sequences in our genome and in that of chimps. Does this mean that all are beneficial? "Our analysis revealed that retroviral sequences in the human genome encode tens-of-thousands of active promoters; transcribed ERV sequences correspond to 1.16% of the human genome sequence ... and PET tags that capture transcripts initiated from ERVs cover 22.4% of the genome."("Ancient Retroviruses Spurred Evolution Of Gene Regulatory Networks In Humans And Other Primates," ScienceDaily, University of California - Santa Cruz, Nov. 15, 2007.) As we can see, it has been discovered that ERVs aid transcription in one fifth of the human genome! "We report that human ERVs actively shape the p53 transcriptional network in a species-specific manner ... At least one ERV insertion likely reshaped the transcriptional landscape of its surrounding genomic area and was instrumental in creating a new gene that became part of the human-specific p53 regulatory network ... We discovered a unique distribution pattern of p53 sites within repetitive sequences of the human genome, and several ERV families emerged as being substantially enriched for p53 sites in their LTRs."("Retroviral promoters in the human genome,2008"